How to maintain your weight after the Progen method VLCKD diet?

In addition to our diet programme, Progen weight management offers a complimentary 2 year maintenance program. Once our clients have reached their ideal body weight after the diet, our dietitians will follow up with them for the next two years. As part of this process, clients are given a diet meal plan tailored to their genetic characteristics. This means that they can eat their favourite dishes and still stay at their ideal body weight.

What is a genetic test? What does it do to one’s body?

Your body is run by memory. In order to maintain a state of normality, the body remembers some aspects of itself. Weight is one of the aspects of the body that it remembers. When you’ve been within 60kgs of your ideal weight for a long time, the body automatically adjusts to keep the weight within that range. This applies regardless of whether your food intake increases or decreases. It is a natural bodily process.

As a result of the ProGen Method, you may have lost a remarkable amount of weight, resulting in your body trying to adjust back to its previous weight. Weight loss with the ProGen Method is less likely to be reversed than with a hypocaloric diet, which is a common way of gaining weight after weight loss. Over time, muscle mass is preserved and improved.

In the maintenance phase, we at ProGen Weight Management, help you through a transition from the fat loss stage to a normal balanced diet. Our dietician plans personalised diet plans based on your specific body needs and eating habits.

In all our patients, we propose studying two genes (FABP2 and ADIPOQ). It is best to adapt to a better diet for them when they are in maintenance, so that we can adjust their diet according to their genetic profile.

The choice of each gene and its variant is justified below:

FABP2

Fatty acid binding protein 2 (FABP2) is an intracellular protein expressed only in the small intestine. Carriers of the THr54 allele, even in heterozygosity, have been reported to have twice the affinity for fatty acids than those without the polymorphism (A common change in the genetic code in DNA). Increased flow of dietary fatty acids in the circulation can lead insulin to impaired glucose metabolism and patients with this polymorphism can have insulin resistance, impaired fatty acid oxidation and certain risk of atherosclerosis.

Patients with the Thr54 polymorphism show increased absorption of fatty acids, which is why they require a very low-fat diet. In the maintenance stage, a low fat intake will be recommended.

ADIPOQ

Adiponectin (AdipoQ), abundantly and specifically expressed in adipose tissue, is an adipokine that has a powerful insulin sensitising effect, and it is involved in the regulation of energy homeostasis.

Its circulating levels can be altered by various genetic and environmental factors, as well as for pathological conditions and certain drugs (1).

Several polymorphisms have been described in the ADIPOQ gene. Specifically, the 276G>T substitution (rs1501299) has been associated with low levels of adiponectin in conditions of low fibre intake, insulin resistance, risk of developing Type 2 DM and even risk of coronary heart disease in individuals with diabetes.

The polymorphisms of this gene represent the most frequent cause of insulin resistance and in the maintenance diet it is suggested to do a low sugar diet and high in fibre for a Type 2 DM better control.

 

 

 

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